Russell Jones
@DrRGJonesLab
Immunology/cancer researcher. Chair, Metabolism Dept at @VAInstitute @VAIMetabolism. Former @McGillU @UofT. GRR/YYZ. Opinions are my own
Happy to announce we are recruiting new faculty to join the Metabolism Department at @VAInstitute. A great opportunity to join a dynamic community of scientists in West Michigan studying the metabolic underpinnings of health and disease. Apply here: vai.wd1.myworkdayjobs.com/en-US/VAICaree…
The gut hormone mimetic era in medicine is just getting started nature.com/articles/s4225… erictopol.substack.com/p/the-gut-brai…
If you are in the Midwest and ❤️ metabolism, take note! This year’s Midwest Metabolism meeting will be held jointly with the Translational Research in Mitochondrial Metabolism in Aging and Disease (TRiMAD) conference. The combined 2-day event will be @umichmedicine, with…
Eight children who were at risk of serious genetic conditions have hit all of their developmental milestones after donor mitochondria was used during their IVF conception newscientist.com/article/248858…
I can’t wait to give this exact speech this fall as an assistant coach for my son’s 8U team @ShoresyHockey Shoresy Inspirational Speech youtu.be/syn-o5zO8XE?si… via @YouTube
Patient-centered information for people using GLP-1 medicines @JAMA_current #diabetes #obesity #heartdisease jamanetwork.com/journals/jamai…
New in @NatImmunol! The @DrRGJonesLab at the @VAInstitute identified the prostaglandin I2 (prostacyclin) receptor PTGIR as a cell-intrinsic regulator of #Tcell exhaustion. go.nature.com/4lcyE7i
AAI Members Dr. Michael Dahabieh and Dr. Russell Jones from the @DrRGJonesLab at @VAInstitute found that the immune checkpoint, PTGIR, may be a potential treatment target to re-energize exhausted immune cells to fight #cancer. Learn more: ow.ly/m6Nc50WiHJC.
The prostacyclin receptor PTGIR is a NRF2-dependent regulator of CD8+ T cell exhaustion @NatImmunol @DrRGJonesLab nature.com/articles/s4159… nature.com/articles/s4159… Preview by @AOxenius
Researchers discovered that cytokine IL-17 plays a key role in polyp formation in the digestive tracts of LKB1 mutant mice. This suggests blocking IL-17 with antibodies may slow the growth of LKB1 mutant tumors. @DrRGJonesLab scim.ag/3ZEYPuF